PNASActivinA通过CD3

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01

Activin-AlimitsTh17pathogenicityandautoimmuneneuroinflammationviaCD39andCD73ectonucleotidasesandHif1-α-dependentpathways

PNAS,

Inmultiplesclerosis(MS),Th17cellsarecriticaldriversoftheautoimmunecentralnervoussystem(CNS)inflammationanddemyelination.Th17cellsexhibitfunctionalheterogeneityfosteringbothpathogenicandnonpathogenic,tissue-protectivefunctions.Still,thefactorsthatcontrolTh17pathogenicityremainin

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